Name: Mitochondrial Oxidative Phosphorylation: Nuclear-Encoded Genes, Enzyme Regulation, and Pathophysiology (en Inglés)
SKU: 60171174
Price: 195.69 USD
Availability: InStock
Author: Kadenbach, Bernhard
ISBN: 9781493901562

portada Mitochondrial Oxidative Phosphorylation: Nuclear-Encoded Genes, Enzyme Regulation, and Pathophysiology (en Inglés)

Mitochondrial Oxidative Phosphorylation: Nuclear-Encoded Genes, Enzyme Regulation, and Pathophysiology (en Inglés)

Formato

Libro Físico

Editorial

Springer

Autor

Kadenbach, Bernhard

Idioma

Inglés

N° páginas

372

Encuadernación

Tapa Blanda

Dimensiones

23.4x15.6x2 cm

Peso

0.54 kg.

ISBN13

9781493901562

Categorías

Genética médica
Bioquímica
Biología molecular

Mitochondrial Oxidative Phosphorylation: Nuclear-Encoded Genes, Enzyme Regulation, and Pathophysiology (en Inglés)

Kadenbach, Bernhard (Autor) · Springer · Tapa Blanda

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Reseña del libro "Mitochondrial Oxidative Phosphorylation: Nuclear-Encoded Genes, Enzyme Regulation, and Pathophysiology (en Inglés)"

This book will describe the nuclear encoded genes and their expressed proteins of mitochondrial oxidative phosphorylation. Most of these genes occur in eukaryotic cells, but not in bacteria or archaea. The main function of mitochondria, the synthesis of ATP, is performed at subunits of proton pumps (complexes I, III, IV and V), which are encoded on mitochondrial DNA. The nuclear encoded subunits have mostly a regulatory function. However, the specific physiological functions of the nuclear encoded subunits of complexes I, III, IV, and V are mostly unknown. New data indicates that they are essential for life of higher organisms, which is characterized by an adult life without cell division (postmeiotic stage) in most tissues, after the juvenile growth. For complex IV (cytochrome c oxidase) some of these subunits occur in tissue-specific (subunits IV, VIa, VIb, VIIa, VIII), developmental-specific (subunits IV, VIa, and VIIa) as well as species-specific isoforms. Defective genes of some subunits were shown to induce mitochondrial diseases. Mitochondrial genes and human diseases will also be covered.